The GluN1, GluN2A, and GluN2B pre-M1 linker is intolerant to genetic
Por um escritor misterioso
Descrição
Cross-subunit interactions that stabilize open states mediate
Structure, Function, and Pharmacology of Glutamate Receptor Ion
Distinct roles of GRIN2A and GRIN2B variants in neurological
Molecular Mechanism of Disease-Associated Mutations in the Pre-M1
GluN2A and GluN2B NMDA receptors use distinct allosteric routes
The role of NMDA receptor and neuroligin rare variants in synaptic
GluN2A-L550A and GluN2A-F553A significantly alter single channel
Regulation of NMDA glutamate receptor functions by the GluN2
Cross-subunit interactions that stabilize open states mediate
Structure, Function, and Pharmacology of Glutamate Receptor Ion
Ionotropic GABA and Glutamate Receptor Mutations and Human
Frontiers Surface Expression, Function, and Pharmacology of
Distinct GluN1 and GluN2 Structural Determinants for Subunit
From bedside‐to‐bench: What disease‐associated variants are
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